Valvular Heart Disease
Clinical overview and exam mastery guide covering aortic and mitral valve disease, drug support strategies, intervention timing, and prosthetic valve anticoagulation.
Valve Lesion Pattern
Medical therapy supports symptoms; intervention is definitive in advanced lesions.
1. What Is Valvular Heart Disease?
Valvular heart disease (VHD) is dysfunction of one or more cardiac valves resulting in stenosis (narrowing) or regurgitation (backflow). The most tested lesions involve aortic and mitral valves.
2. Major Valve Disorders
Aortic Lesions
- Aortic stenosis (AS)
- Aortic regurgitation (AR)
Mitral Lesions
- Mitral regurgitation (MR)
- Mitral stenosis (MS)
3. Aortic Stenosis (AS)
Pathophysiology
- Calcific degeneration or congenital bicuspid valve
- LV outflow obstruction and increased afterload
- Left ventricular hypertrophy
Classic Symptoms
- Syncope
- Angina
- Dyspnea (heart failure)
SAD is the classic severe AS triad.
4. Aortic Regurgitation (AR)
Incomplete valve closure causes diastolic backflow into the LV, leading to chronic volume overload and LV dilation.
| Therapy | MOA | Side Effects | Contraindications |
|---|---|---|---|
| ACE inhibitors | Reduce angiotensin II and systemic vascular resistance (afterload reduction) | Cough, hyperkalemia, renal impairment | Pregnancy, bilateral renal artery stenosis |
5. Mitral Regurgitation (MR)
Backflow into the left atrium causes volume overload, left atrial enlargement, and increased risk of atrial fibrillation.
Management Priorities
- Valve repair is preferred for suitable symptomatic patients
- Afterload reduction support (e.g., ACE inhibitors)
- Diuretics for congestion symptom control
Loop Diuretics
- MOA: inhibit Na-K-2Cl transporter in loop of Henle
- Effect: reduce volume overload symptoms
- Side effects: hypokalemia, dehydration, hypotension
- Role: symptom control only (not definitive lesion correction)
6. Mitral Stenosis (MS)
Often rheumatic in origin. Obstructed LV filling raises left atrial pressure, causes pulmonary congestion, and increases AF/thromboembolic risk.
Beta Blockers
- MOA: slow heart rate and increase diastolic filling time
- Side effects: bradycardia, fatigue
Anticoagulation (If AF)
- Warfarin preferred in rheumatic MS with AF
- Goal: reduce systemic embolic stroke risk
7. Prosthetic Valves
Mechanical Valves
- Durable option
- Requires lifelong warfarin anticoagulation
Bioprosthetic Valves
- Less durable
- Shorter anticoagulation duration in many cases
| Drug | MOA | Monitoring | Contraindications / Traps |
|---|---|---|---|
| Warfarin (mechanical valve) | Inhibits vitamin K-dependent factors II, VII, IX, X | INR typically 2.5 to 3.5 (valve dependent) | Pregnancy contraindication; DOACs contraindicated in mechanical valves |
8. Infective Endocarditis Prophylaxis
Who Needs Prophylaxis?
- Prosthetic valve patients
- Prior infective endocarditis
- Certain congenital heart diseases
Procedure Context
- Before high-risk dental procedures with gingival manipulation
Amoxicillin (First-line)
- MOA: beta-lactam, inhibits bacterial cell wall synthesis
- Use: prophylaxis in high-risk groups only
- Major side effects: hypersensitivity rash, GI upset
- Contraindication: serious beta-lactam allergy
Management Recap Drill
Visual Algorithm Placeholder
[Insert Valvular Disease Diagnostic and Treatment Flowchart Here During UI Integration]
Guideline References (Management)
ACC/AHA Guideline: Valvular Heart Disease
https://www.acc.org/guidelinesHigh-Yield Scope
- Timing of surgery/intervention
- TAVR candidacy principles
- Prosthetic valve anticoagulation
9. Common Exam Traps
10. Quick Revision Summary
Core Memory Anchors
- AS: pressure overload
- AR: volume overload
- MR: left atrial dilation and AF tendency
- MS: often rheumatic
- Mechanical valves: lifelong warfarin
Practice Questions Placeholder
- Topic: Valvular Heart Disease
- Subtopics: aortic stenosis, aortic regurgitation, mitral regurgitation, mitral stenosis, prosthetic valves, endocarditis prophylaxis