Thyroid Disorders
Clinical overview and exam mastery guide for hypothyroidism, hyperthyroidism, thyroid storm sequence, and levothyroxine/antithyroid drug strategy.
TSH Axis and Drug Targets
Drug sequencing is especially critical in thyroid storm.
1. Thyroid Physiology (Foundation First)
Thyroid gland produces T4 and T3 (active form). Regulation follows hypothalamic TRH to pituitary TSH to thyroid hormone release, with T3/T4 negative feedback on TSH.
2. Hypothyroidism
Common Causes
- Hashimoto thyroiditis (most common)
- Post-thyroidectomy or radioiodine
- Iodine deficiency
- Drug-related (e.g., amiodarone)
Clinical Features
- Weight gain, fatigue, cold intolerance
- Constipation, bradycardia, dry skin
- Severe emergency: myxedema coma
3. Management of Hypothyroidism
Levothyroxine (T4)
- MOA: synthetic T4 replacement converted peripherally to T3
- Dosing: take on empty stomach; separate calcium/iron by at least 4 hours
- Monitoring: recheck TSH every 6 to 8 weeks after dose changes
- Over-replacement effects: tachycardia, arrhythmias, long-term bone loss risk
- Contra/caution: untreated adrenal insufficiency; cautious titration in elderly/CAD
4. Myxedema Coma (Emergency)
Typical Features
- Hypothermia
- Bradycardia
- Altered mental status
Treatment Core
- IV levothyroxine
- IV hydrocortisone until adrenal insufficiency is excluded
- Supportive ICU-level care
5. Hyperthyroidism
Causes
- Graves disease (most common)
- Toxic multinodular goiter
- Thyroiditis
- Amiodarone-associated thyroid dysfunction
Clinical Features
- Weight loss, heat intolerance, tremor
- Palpitations, anxiety, diarrhea
6. Management of Hyperthyroidism
Core options: antithyroid drugs, radioactive iodine, or surgery.
| Drug/Class | MOA | Major Side Effects | Contraindications / Notes |
|---|---|---|---|
| Methimazole (first-line) | Inhibits thyroid peroxidase and blocks T3/T4 synthesis | Rash, agranulocytosis, hepatotoxicity | Avoid in first-trimester pregnancy (use PTU) |
| Propylthiouracil (PTU) | Inhibits thyroid hormone synthesis and peripheral T4 to T3 conversion | Hepatotoxicity (greater severity risk), agranulocytosis | Preferred in first trimester and thyroid storm contexts |
| Beta blockers (e.g., propranolol) | Beta blockade reduces adrenergic symptoms; propranolol also lowers T4 to T3 conversion | Bradycardia, fatigue, hypotension | Symptom control in most hyperthyroid presentations |
7. Thyroid Storm (Life-Threatening)
Features
- High fever
- Tachycardia
- Delirium/encephalopathy
- Heart failure risk
Critical Exam Sequence
- 1. PTU
- 2. Iodine (after PTU)
- 3. Beta blocker
- 4. Steroid
8. Subclinical Thyroid Disease
Defined by abnormal TSH with normal circulating T3/T4. Treatment decisions depend on TSH magnitude, symptoms, age, and comorbidity profile.
Management Recap Drill
Visual Algorithm Placeholder
[Insert Thyroid Disorder Diagnosis and Treatment Algorithm Here During UI Integration]
Guideline References (Management)
American Thyroid Association Guidelines
https://www.thyroid.orgGuideline Scope
- Levothyroxine dosing and monitoring
- Hyperthyroidism treatment pathways
- Thyroid storm protocol
9. Common Exam Traps
10. Quick Revision Summary
Must Remember
- TSH guides screening and dose adjustment
- Hypothyroidism treatment is levothyroxine replacement
- Hyperthyroidism usually starts with methimazole
- Thyroid storm starts with PTU before iodine
- Beta blockers control adrenergic symptom burden
Practice Questions Placeholder
- Topic: Thyroid Disorders
- Subtopics: hypothyroidism, hyperthyroidism, methimazole, PTU, thyroid storm, levothyroxine monitoring